Activating Notch1 mutations in mouse models of T-ALL.

نویسندگان

  • Jennifer O'Neil
  • Jennifer Calvo
  • Keith McKenna
  • Veena Krishnamoorthy
  • Jon C Aster
  • Craig H Bassing
  • Frederick W Alt
  • Michelle Kelliher
  • A Thomas Look
چکیده

Recent studies have demonstrated that most patients with T-cell acute lymphocytic leukemia (T-ALL) have activating mutations in NOTCH1. We sought to determine whether these mutations are also acquired in mouse models of T-ALL. We sequenced the heterodimerization domain and the PEST domain of Notch1 in our mouse model of TAL1-induced leukemia and found that 74% of the tumors harbor activating mutations in Notch1. Cell lines derived from these tumors undergo G(0)/G(1) arrest and apoptosis when treated with a gamma-secretase inhibitor. In addition, we found activating Notch1 mutations in 31% of thymic lymphomas that occur in mice deficient for various combinations of the H2AX, Tp53, and Rag2 genes. Thus, Notch1 mutations are often acquired as a part of the molecular pathogenesis of T-ALLs that develop in mice with known predisposing genetic alterations.

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عنوان ژورنال:
  • Blood

دوره 107 2  شماره 

صفحات  -

تاریخ انتشار 2006